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In infants, untreated deficiency may result in neurological impairment and anemia

Some individuals inherit a genetic characteristic known as the hungry gut phenotype, resulting in reduced levels of satiety after eating and faster gastric emptying compared to the general population.1 These individuals tend to respond more favorably when treated with glucagon-like peptide 1 (GLP-1) agonists and experience higher levels of weight loss with semaglutide, a GLP-1 agonist

E., Roffe, C., Smith, C

Eating foods that support GLP-1s can reduce the chances of side effects and improve health outcomes

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